Awesome Article thrown together by my brother Elmar!
Garlic (Allium sativum) is reputed to offer protection against (unless stated otherwise):
* Stroke
* Coronary thrombosis ( a form of thrombosis affecting coronary circulation; thrombosis is the formation of a blood clot (thrombus) inside a blood vessel, obstructing the flow of blood through the circulatory system)
* Atherosclerosis (a condition in which an artery wall thickens as the result of a build-up of fatty materials such as cholesterol) — By the ability to inhibit platelet aggregation
* Garlic is FOR Inhibition of Microbial Growth
* FOR Lowering Blood pressure
* FOR Lowering Cholesterol levels by: inhibiting HMG-CoA (enzyme HMG-CoA produces cholesterol)
* FOR fighting cancer/tumor (S-allylcysteine and garlic’s organosulfur compounds posses anti-carcinogenic properties that are able to inhibit cancer cell growth).
- carcinogenic = any substance or agent that tends to produce a cancer.
2 VERY IMPORTANT ingredients: Allicin and Ajoene
1) Allicin:
* Increases Blood levels of two antioxidant enzymes ( catalase & glutathione peroxidase)
* Retards the aging process in Liver (by inhibiting lipid peroxidation in the liver)
* Prevents oxidant injury in vascular endothelial cells.
* Fights Tumors by: Increasing the apoptosis (self-programmed death of a cell) rate of hepatocellular (pertaining to or affecting liver cells) tumor cells.
- Study of rate of apoptosis in rats shows:
Group 1: Rats without being given anything had an apoptosis rate of only 2.5%.
Group 2: Rats given ADM(adriamycin- chemotherapy drug) shown an apoptosis rate of 23.6% (±3.1%)
Group 3: Rats given Allicin(high amount) shown an apoptosis rate of 29.5% (± 3.4%)
Group 4: Rats given Allicin(high amount) + ADM shown an apoptosis rate of
36.9% (±4.2 %)
2) Ajoene (Sulfur-Containing Compound Derived from Garlic):
* Exhibits broadspectrum antimicrobial activity
* Blocks HIV-mediated syncytia formation: possible approach to ‘anti-adhesion’ therapy of AIDS
- Source: http://gateway.nlm.nih.gov/MeetingAbstracts/ma?f=102201963.html
* Inhibits platelet deposition on severely damaged vessel wall in an in vivo porcine experimental model
- Source: http://www.ncbi.nlm.nih.gov/pubmed/7992235
* Potent inhibitor of platelet aggregation (antithrombotic activity): by inhibiting the main receptor on platelets)




